د.وحده

Practical of Cell Injury Third Year 3rd October 2016

Coagulative necrosis- Kidney

Coagulative necrosis-Spleen

Coagulative necrosis (Myocardial infarction in the heart)

Liquefactive necrosis- Brain infarction

Caseous necrosis- involving hilar lymph node & is elicited grossly as cheesy like material


Fat necrosis of the mesentery: chalky white patches on the surface of mesentery due to enzymatic digestion of mesenteric fat secondary to acute pancreatitis

Fatty necrosis-Acute pancreatitis

Artery, fibrinoid necrosis

Apoptotic body appears as dense eosinophilic core

Dry gangrene -Ischemia

Wet gangrene –D.M

Endometrial hyperplasia is an example of hormone-induced hyperplasia due to hyperestrogenism.

Endometrial hyperplasia-there is hyperplasia of the both glandular & stromal elements.

Hypertrophic cardiomyopathy is an example of pathological hypertrophy due to increase demand, this ultimately results in increase in the size of the organ

hypertrophy of cardiac muscle in response to increased demand

Squamous metaplasia of bronchial epithelium

Fatty change of the liver with vacuolated hepatocytes.

Lipofuscin pigment-liver wear & tear pigments

Hemosiderin pigment in the alveolar macrophages

Carbon particles- Lung & LN

dystrophic calcification affecting aortic valveAortic valve, dystrophic calcification

Dystrophic calcification of a vessel at the base of gastric ulcer

Dystrophic calcification of dead parasite in the lung

Metastatic calcification in the lung