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Structure of local anesthesia

Ester:

Amide:

Example:

Exception:

Benzocaine, which lacks a substituted amino group
R —COO—R —N

R —NHCO—R —N

1
2

R
R
3
4
2
1


R
R
3
4
H N—
—COO—(CH ) —N

2
2
2

C H
2
5

C H
2
5
R — Lipophilic aromatic residue.

R — Aliphatic intermediate connector.


R , R — Alkyl groups, occasionally
H. Constitute with N the hydrophilic
terminus.
1
2
3
4

PH and local anesthesia

Dissociation constant PKa:
pH at which 50% of drug present in free base form and 50% in cationic form (water soluble)
Most local anesthetic Pka(7-9)
How can local anesthesia cross nerve membrane?
Local anesthetic solution prepared as weak acid form at 4-5 pH to prevent precipitation of free base in neutral solution, thus it present as cataionic form that must converted to base form to be lipid soluble and cross cell membrane


local ansthesia

Buffering capacity

Local anesthetic agent after injection and with function of plasma buffering will dissociate into free base form and cataionic form
Free base form cross the cell membrane
After crossing the membrane an other dissociation occur and cataionc form resulted will bind the receptor


Onset (induction time)
• Time from injection of local anesthesia to the sign of adequate surgical anesthesia achieved
• Factors affect onset (induction time)
• Concentration
• pH
• PKa
• Anatomical barrier
• Lipid solubility

Recovery

Time from early sign appeared to the complete loss of all effects of drug occur
This results from reduced concentration of drug with no binding with the receptors

Recovery time

Concentration gradient depleted by:
Dilution by interstitial fluid
Action of capillary and lymph
Absorption by other tissue
Hydrolysis of ester

Extraanuronal


Diffusion from intranuronal tissue (mantle fiber)

Duration

Time from induction to complete recovery from local anesthesia
It depend on:
• Protein binding
• Vasoactivity
• concentration
• Vascularity of the site
Recovery slower than induction ?
Protein binding capacity


local ansthesia

Principle of reinjection

Profound anesthesia:
Increase concentration gradient to mantle fiber then to the core fiber

• Failure ??

• Edema
• Localized hemorrhage
• Clot formation
• Reduced pH (poor buffer capacity)
• hypernatremia
After reinjection in prolonged procedure 2 situation may occur


Progression of local anestheticfunction
Dull pain
Temprature
Sharp pain
Touch
Deep pressure
Proprioception
Motor function

H. W.

Why infected area presents a poor site for action of local anesthesia




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