background image

 

 

 Vascular Surgery

 

Lec :   3

 

Venous System :

 

Varicose Veins :-

  defined as tortuous dilated veins which affect 

5% of adult population.  M > F but most Pt.s seek for Rx. Are F. 
there is +ve family history for inheritance through  FOXC2 gene.

 

V.V develops due to defect in connective tissue and smooth 
muscles in vein wall leading to secondary incompetence in the 
valves , other predisposing factors are: -

 

1.DVT      2.Pregnancy         3.Pelvic tumors         4.Age

 

5.Weight and Hight- heavy weight and tall pts. are more liable.

 

6.Diet – Low fiber diet leads to chronic constipation which → V.Vs

 

7.side - left > right         8. Occupation with long standing period.

 
 

 C.F :- 1. Ache in the vein after prolonged standing.

 

2. Ankle swelling , itching , bleeding , superficial thrombophlibitis, 
ulceration.    3.Tortous dilated veins in the subcutaneous tissue

 

  4. varicosities in the thigh are indicative of long SV incompetence 
while varicosities on the back of the leg are suggestive of short SV 
incompetance 

 

5. Cough thrill –a vibration can be felt by gentle palpation through 
the fingers over sapheno-femoral junction while the pt. is 
coughing caused by turbulent backflow with in the varicosity.

 

6. Torniquet test  – when tourniquet is applied round the upper 
thigh after elevation of the limb and emptying of the veins can 
control all lower limb varicosities indicating  sapheno-femoral 
junction incompetence , release of  tourniquet leads to rapid refill 
of varicosities confirm the Dx. Greater SV varicosity represent 80% 
of cases.

 

Test for Lessor SV. By tourniquet placed below Knee to test 
Sapheno-popliteal junction incompetence.

 
 


background image

 

 

Duplex u/s imaging – the probe of Duplex scanner contains 
multiple emitting and receiving crystals , this allow the vein to be 
seen as a black void in subcutaneous and deep tissues. Directional 
flow can be seen as a coulor image super-imposed on grey scale 
image of the vessel , visible venous flow can be seen when 
augmented  by calf squeeze , the duplex image allow the vein to 
be traced to its termination while compression and relaxation can 
demonstrate the presence of retrograde flow( reflux).

 
 

Mx – indication for intervention / 

 

 1.cosmotic appearance esp. for female.   2.Reccurent episode of 
bleeding.    3.development of skin changes.

 

a.if varicose vein is secondary to DVT or elderly Pt. who doesnot 
not seek surgery Rx. done By use of below Knee Class 2 Elastic  
compression stockings with Ankle pressuer 30mm Hg decrease to 
15 mm Hg at knee level.

 

b. Injection seclerotherapy using Sodium Tetredecyl Sulphate is 
injected directly into the superficial veins to destroy the lipid 
memberane of endothelial cells which will shed leading to fibrosis 
and obliteration of vein with local compression on injected veins , 
it is useful for minor varicosities and recurrent cases in calf and 
lower leg.

 

c. Surgery – done by ligation of the communication point i.e 
sapheno-femoral or Saphenopopliteal junctions and remove the 
major part of incompetent trunk  using Babcocks intraluminal 
stripper then small tributaries are removed individually by making 
minor cut downs and avulse them out from under the skin.

 

 

D.V

.

T :- 

is the formation of semisolid coagulum with in the 

flowing blood in the venous system , it carries high risk of 
Pulmonary Embolism and Sudden death.

 

 

Etiology -  1. Change in vessel wall ( endothelial damge).

 


background image

 

 

2. Stasis leads to decrease blood flow through the vein.

 

3. increase blood viscosity

 

Risk factors for DVT-   1. Pt. factors   a. age    b.obesity       
c.varicosity       d.immobility        e.pregnancy     f.puerperium    
g.high dose Estrogene Rx        h.previous DVT.

 

 

2. Disease or surgery- a.Trauma or surgery esp. pelvic , hip.

 

b. Malignancy eps. Pelvic or abdominal.      C. Heart failure

 

d. Recent MI         e. Lower limb paralysis    f. infection

 

      Pathology :- the thrombus usually develop in the soleal veins of 
calf initially as platelet aggregate then fibrin and red cells form a 
mesh until the lumen of the vein occludes , the thrombus extend 
up to next large vein branch and may break off and pass through 
the large veins of lower limb and IVC and through the right side of 
the heart  and embolise to the pulmonary arteries of the lung as  
P.E. , this may totally occlude perfusion to all or part of one or 
both lungs leading to acute right side heart failure with sudden 
collapse and death.

 

 

 

Dx : – 

 

1. Pain and Swelling in the calf of one lower limb .

 

2. pt. may present from start with ss of P.E e.g Dyspnea , 
hemoptasis , pleuretic chest pain.

 

O/E – 1. Mild pitting edema of ankle     2. Dilated superficial veins.

 

3. stiff calf      4.Low grade fever.  5. Tenderness over the course of 
deep veins.

 

heart sound 

  

nd

1.cyanosis     2. Dyspnea  3.fixed splitting of 2

 

-

P.E

 

4.pleural rub.

 

DVT Dx confirmed by Duplex U/S imaging which shows filling 
defects in the flow and lack of copressibility.

 

P.E Dx confirmed by CT scan of Chest which shows filling defect in 
the pulmonary arteries.

 

DDx of DVT is :-  1.ruptuerd backers cyst   2.calf muscle hematoma

 


background image

 

 

3. thrombosed popliteal aneurysm   4.arterial ischemia.

 

 

Prophylaxis

:-  pt. who are being admitted for surgery are classified to 

Low , moderate and high  risk group.

 

1.Low risk pt.-  are young with minor illness who will undergo opt. which 
last 30 min or less.

 

2. Moderate risk pt. -  age >40 yr. or have debilitating illness and will 
undergo major surgery.

 

3. High risk pt. – age >40 yr , have serious medical illness e.g CVA or MI , 
undergo major surgery with previous Hx of thromboembolism or 
malignant disease.

 

Prophylactic methods which can be used are as following

 

1.good post-opt I.V hydration is simple method as the pt. will stay fasting 
for about 8 hours pre-opt together with internal body response which 
increase blood coagulability post-opt. and the inability of pt. to drink oral 
fluid in immediate post-opt. period all this will increase blood viscosity and 
make the pt highly susceptible to develop thromboembolism , therefore ; 
good I.V fluid Mx is very important and it breaks the vicious circle of stasis 
and increase blood viscosity

 

2.encourage the pt. to move his or her feet in the bed aginst some 
resistance this will make the calf muscles to contract and squeeze the 
blood from the venous sinusoids preventing clot formation

 

3. for pt.s who cannot move  post-opt. e.g  limb fracture we can use 
graduated elastic compression stockings and external pneumatic 
compression devices

 

4.Low-molecular weight heparin can be given subcutaneously 50 mg/Kg 
body Wt. as single or in 2 divided doses 

 

 

Heparin I.V infusion 1000U/hr. together with oral Warfarine 

 

start with

 

Rx 

three days then heparin stopped and Pt. kept on warfarine whose 

 

st

for 1
dose adjusted according to PT , INR ratio which must be between 2-3 , the 
pt must be kept in bed with minimum movement for 1 Wk to prevent clot 
detachment since the clot inside the vein is loosely adherent to vein wall 

wk and easily can detach and cause pulmonary Embolism. 

 

st

in the 1

 

P.E requires 1. Thrombolytic Rx       2.Cardiac support    3.I.V fluid

 

4. Pulmonary Embolectomy Through Median Sternotomy and 
Cardiopulmonary bypass.  

 


background image

 

 

 
 

   

 




رفعت المحاضرة من قبل: Ahmed 95
المشاهدات: لقد قام 17 عضواً و 170 زائراً بقراءة هذه المحاضرة








تسجيل دخول

أو
عبر الحساب الاعتيادي
الرجاء كتابة البريد الالكتروني بشكل صحيح
الرجاء كتابة كلمة المرور
لست عضواً في موقع محاضراتي؟
اضغط هنا للتسجيل