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Fetal monitoring during lobar


Fetal monitoring during lobar


By
Dr. Huda Adnan Alrabaiy

C.A.B.O.G

Fetal monitoring during lobar

Fetal monitoring during labour


The objective of fetal monitoring during labour is the prediction & diagnosis of fetal asphyxia before fetal / newborn morbidity with particular reference to brain damage has occurred .
- Fetal asphyxia :
Is defined as a condition of impaired blood gas exchange leading , if it persists , to progressive hypoxemia & hypercapnia .

Fetal monitoring :

Is performed in all labours ,low-risk labour should be monitored by intermittent auscultation by ear ( pinard ) or by using hand-held Doppler devices .
- High-risk labours employ continuous electronic FHR monitoring , either ways the rate is entered on the partogram ( every 15 min – 1/2 hr ) .
So on admission ask your self ?
Are there any risk factors present ? e.g.


1. Maternal problems :
1. previous C/S . 6. DM ( Diabetes )
2. PET ( pre-eclampsia ) 7. APH ( antepartum heamorrhge)
3. Postterm pregnancy .
4. Induced labour 8. Maternal Fever .
5. Oxytocin use . 9. Epidural analgesia .

2. Fetal problem :

1. IUGR ( intrauterine growth restriction )
2. Oligohydramnias .
3. Meconium stained – liquor .
4. Multiple pregnancy .
5. Abnormal Doppler arterial velocimetry .
If the answer is =No intermittent Auscultation .
=Yes( CTG )
( continuous cardiotocography )

Intermittent Auscultation of FHR :

1. For 6o sec. after a contraction .
2. Every 15 – 30 min in 1st stage .
if abnormal FHR:shift to contineous CTG.
3. Every 5 min in 2nd stage .
if abnormal FHR:shift to contineous CTG.

Contineous CTG :

1. External machine which is a probe that measures uterine contractions and fetal heart rate & both are recorded on a paper and we see the relationship between them & assess the fetal well-being .
- The female should be Lt- lateral or semi-recumbent position to avoid vena-cava compression , ( 2 transducers for : FHR + tocodynometer ) .
- Recordings are made for at least 30 mins .

2. Internal machine : More invasive , but more accurate , the probe is placed inside the amniotic sac to assess uterine contractions , but they need rupture of membranes , fetal scalp electrode for fetal ECG , it may cause ascending infection to the fetus .
Four parameters should be evaluated in each record :
1. Basal FHR , 2. variability 3. accelerations . 4. decelerations .

* Factors that cause fetal distress during labour :

I ) Maternal causes :
1. Hypotension
2. sever maternal anemia
3. cardiac disease .
4. seizures
5. pulmonary diseases
6. hypertension .
II ) Fetal causes :
1. anemia ( as in Rh – Iso immunization )
2. Infection .
3. Twin to twin transfusion .


III ) Uterine causes :
1. Tetanic contractions of the uterus .
2. Hyperstimulation : The commonest cause , is iatrogenic by uncontrolled oxytocin .

IV ) Umbilical cord causes :

1. Single umbilical artery blood flow to the fetus .
2. Vasa – praevia .
3. Short-cord

4.placental Haematoma .

5. True-knots in umblical cord
6. Prolapse of the cord .

V ) Placental causes :

1. Infection .
2. placenta abruption .
3. Post mature placenta .
(reduce functioning due to aging ) .

Abnormalities of fetal heart monitering:


1. Brady cardia : Defined as a decrease in the baseline FHR to < 110 bpm .
- Possible causes : 1. Fetal hypoxia . ( as late sign )
2. Medications ( Narcotics )
3. Epidural anesthesia .
4. Oxytocin ( pitocin )
5. Maternal hypotension .
6. Cord prolapse .

2. Tachy cardia : Baseline FH > 160 bpm.

Possible causes :
1. Fetal hypoxia ( as early sign )
2. Medications ( ex. terbutaline )
3. Prematurity .
4. Maternal anxiety .
5. Maternal fever .
6. Fetal infection .
7. Thyrotoxicosis .

3. Absence of Variability :

variability is the most reliable sign of fetal well being . Is the normal irregular changes & fluctuations in the FHR around baseline rate , so the baseline rate should vary by at least ( 10 – 15 ) beats over a period of one minute , so the tracing appears as a jagged , rather than a smooth .


- Factors affect variability
1. Hypoxia .
2. Fetal infection .
3. drugs that suppress the CNS ( opoid , hypnotics )
4. extreme premature
5. Mg-sulfate use
6. Congenital heart block .
7. Fetal sleep (the baby speels in 20 – 40 minute cycles)

4.Absence of accelerations

Acceleration defined as a transient increase in heart rate of greater than 15 bpm for at least 15 seconds ( the 15 x 15 rule )
Accelerations are a reassuring sign as they show fetal responsiveness & integrity of the mechanisms controlling the heart .
5.Decelerations : Transient fall in FHR > 15 beats lasting > 15 second .

Types of deceleration :

1.Early deceleration : Begins at or after the onset of a contraction & returns to the baseline rate by the time the contraction has finished & produces a mirror image of the contraction .
- it not sign of fetal problem , It's due to vagal nerve stimulation .
2. Late decelerations : Are transitory reduction in FHR caused by utero-placenta insufficiency .
- Begins after the onset of the peak or middle of the contraction & ends after the contraction .

- The presence of late deceleration , justify secondary testing .

Variable deceleration
In which there is decrease in fetal heart rate not related to uterine contractions. - This results from direct myocardiac depression secondary to hypoxemia .


Management of fetal heart rate pattern :

1- Early deceleration : The majority doesn’t fall below 100 bpm , usually is associated with uncomplicated labour , If falls < 100 bpm & is repetitive so pervaginal examination to exclude cord prolapse .
2- Late deceleration : If oxytocin in use so stopped .When the pattern has been corrected & after appropriate resolution , oxytocin may be restarted , start O2 5-6 L/min by tight-fitting O2 mask . , change position to left lateral position , increase rate of electrolytes containing fluid , assess fetal acid-base status by fetal stimulation test , if positive, so the pH is > 7.25 , or by fetal blood sampling ( for pH , lactate level )

Management of abnormal fetal heart

1. Stop oxytocin 2. O2 3. Lateral position .
4. pervaginal examination to exclude cord prolapse and to determine the progress of labour .
5. If uterine activity excess, use tocolytic such as torbutaline or salbutamol.
6. If not corrected consider delivery .

Secondary Tests of fetal well-being

1. Vibroacoustic stimulation :
An acceleration evoked by vibroacoustic stimulation immediately prior to scalp sampling was never associated with a pH of < 7.25 .
- It can reduce the need for scalp sampling by up to 50 % .
2. Fetal blood sampling :
- The lower limit of normal pH level is 7.20 .
- The base excess should also be measured to distinguish metabolic from respiratory acidosis .
- The amount of blood required is 25 microliter .
3. Scalp stimulation :
That fetuses that respond to scalp sampling with an acceleration almost always have an normal pH .
4. Fetal electrocardiogram : ( ECG )
5. Fetal pulse oximetry .

Objective

The objective of fetal monitoring during labour is the prediction & diagnosis of fetal asphyxia before fetal / newborn morbidity with particular reference to brain damage has occurred .
How we can improve the outcome by intra-uterine resuscitation , and by using the secondary fetal-well-being tests .
Decrease the operative delivery by using secondary fetal-well being tests .
Objectives of partogram are to reduce the use of oxytocin . and to improve the outcome , detect any labour abnormalities and their underlying cause , reduce the operative intervention , and identify the patient at high risk to develop obstructive labour .



Fetal monitoring during lobar


Fetal monitoring during lobar





رفعت المحاضرة من قبل: Mubark Wilkins
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