*
* I. general complications:
Blood loss. Shock. diffuse coagulopathy . respiratory dysfunction. metabolic response.* tetanus. gas gangrene. fat embolism.
* Crush syndrome. (if bulk muscle is crushed it is reflected on the kidney function ,end with renal failure.) Venous thrombosis &pulmonary embolism. In veins of the calf and less in the thigh.* II Local complications
A:early . ( bone ,soft tissue). B:late. (bone ,soft tissue,joint).* Early complications bone
Infection particularly in compound fracture.*
* Vascular injury.
Brachial artery. Popliteal artery.
* Nerve injury.
(neuropraxia, axonotemesis, neurotemesis).* nerve
InjuryAxillary
Shoulder dislocation
Radial
Fracture humerus shaft
Radial or median
Supracondylar humerus
Ulnar
Medial epicondyle
Posterior interosseous
Monteggia fracture dislocation
Sciatic
Hip dislocation
* Visceral injury.
diaphragm (in fracture pelvis).* Compartment syndrome.
Fracture of the arm or the leg can give rise to severe ischaemia even if there is no damage to a major vessel. this lead to reduce in the capillary flow.* Bleeding,edema, inflammation
Increase the interistial pressure Within osteofascial Compartment > 40mmReduce in capillary flow
Muscle ischaemia
Further edema
Greater pressure &greater ischaemia
*
*
* After 12 hours or less necrosis of the nerve,which is capable for regeneration.But muscle once infract can never recovers and is replaced by inelastic fibrous tissue(VOLKMANNS’ ischaemia contractures)
*
* Clinical features
Five Ps. Pain with sever swelling . Parasthesia. Pallor. Paralysis. Pulse less.* Treatment
Removal cast bandage. Fasciotomy >40 mm Hg. <40 mmHg close observation. The wound should be left open and inspected 5 days late to be suture or skin graft.*
*
* Fracture blisters occurred by edema. Plaster sores. Torn muscle fibers.
* Haemarthrosis. Gas gangrene with clostridium M.O. in a dirty wound* Late complicationsbone
avascular necrosis: Due to ischaemia after injury.as fracture neck femur.*
* Delayed union:
1.inadequate blood supply. 2.infection. 3.incorrect splintage. 4.intact fellow bone (as in fracture tibia and fibula).* Non union:
1.too large gap. 2.Soft tissue interposition. 3.Intra articular fractures(scaphoid).*
*
* Mal union: When the fragments join in an unsatisfactory position.
* Growth disturbance damage to the physis may lead to abnormal or arrested growth.*
*
* Late soft tissue complications
Myositis ossificans (heterotopic ossification in muscle as after elbow injury ). Bed sores* Tendinitis: (tibilalis posterior in ankle fracture). Tendon rupture: (extensor pollicis longus in colles).
* Nerve compression: as in radial palsy followed faulty use of crutches. Nerve entrapment: ( median nerve following injuries around wrist).
* Volkmann's’ contracture
** Late jointscomplication
Sudecks’ atrophy.(painful osteoporosis of the hand ,reflex sympathetic dystrophy ).*
Osteoarthritis. (fracture involving a joint may severely damage the articular cartilage).
* Instability . (muscle weakness, gun shot with bone loss, recurrent dislocation) Joint stiffness. due to oedema and fibrosis of the capsule.
*
* Osteogesis imperfecta.Pagets’ disease.Chronic infection.Solitary bone cyst.
**
* Aneurysmal bone cyst. Osteosarcoma. Ewing's sarcoma. Metastatic tumors
* Clinical features of pathologicalFracture develop spontaneously or after trivial injury.
* Examinations:
general &Local.* Investigations:
x-ray, blood, urine, scanning, biopsy.*
*